is that related to mammals only?
www.health.harvard.edu/staying-healthy/red-meat-tmao-and-your-heart
LP(a) lipoprotein(a)
Keto for PD, Some Evidence
…What are the benefits of Ketosis?
Achieving a state of ketosis can have many benefits from treating chronic illnesses to optimizing performance. While the benefits are well documented, the underlying mechanism of action is not entirely known. The diet seems to enhance the ability of mitochondria, the power plants of our cells, to deliver our bodies’ energy needs in a manner that reduces inflammation and oxidative stress. Through optimizing the way our body uses energy, we fortify our bodies’ ability to take on the ever-growing stressors of our modern way of living.
some results:
charliefoundation.org/keto-for-parkinsons/
Management of EDTA Chelation Therapy
Regulated Vitamine D level to prevent calcification
stop and reverse PD, the state of the art
Ketoprofen, anti inflammatory drug reduce/reverse Lymphedema sysmptoms
Vitamin D
does vit D cause calcification of arteries?
according to one article, it could in some settings, not clear when, many other sources point out the benefits of vit d
Vitamin D in Vascular Calcification: A Double-Edged Sword?
#q
How much sun exposure is needed for vit D instead of supplements?
answer from https://www.healthline.com/nutrition/vitamin-d-from-sun#time-of-day
At noon, the sun is at its highest point, and its UVB rays are most intense. That means you need less time in the sun to make sufficient vitamin D (5Trusted Source).
Many studies also show that the body is most efficient at making vitamin D at noon (6Trusted Source, 7Trusted Source).
For example, in the UK, 13 minutes of midday sunlight exposure during summer three times per week is enough to maintain healthy levels among Caucasian adults (5Trusted Source).
Another study found that 30 minutes ttof midday summer sun exposure in Oslo, Norway was equivalent to consuming 10,000–20,000 IU of vitamin D (8Trusted Source).
The commonly recommended daily dose of vitamin D is 600 IU (15 mcg) (3).
Vitamin D is critical for brain health…
Reductive stress – to much antioxidants
Stop and reverse PD
Probiotics for PD
Do brain-training games really work?
New scientific evidence challenges the alleged cognitive benefits of brain-training games. Exercise and sleep may be better ways to boost cognition.
electrolytes including sodium, calcium, potassium, magnesium, and chloride negativly asssociated with Dyskinesia in PD
Stem Cell – Repairing the Brain: Cell Replacement Using Stem Cell-Based Technologies – IOS Press
Current approaches to cell replacement therapy in Parkinson’s disease are strongly focused on the dopamine system, with the view that restoring dopaminergic inputs in a localized and physiologic manner will provide superior benefits in terms of effec
Source: Repairing the Brain: Cell Replacement Using Stem Cell-Based Technologies – IOS Press
CBD – Cannabinoids in PD
Fetal tissue implant – Wikipedia
Source: Fetal tissue implant – Wikipedia
…In 1982, seven people in Santa Clara County, California were diagnosed with Parkinsonism after having used MPPP contaminated with MPTP. In 1992, two of the seven patients were successfully treated at Lund University Hospital in Sweden with neural grafts of fetal tissue. One patient, who had been essentially paralyzed, regained enough motor function to ride a bicycle.[1]
Diabetes protocol
High Prevalence of Undiagnosed Insulin Resistance in Non-Diabetic Subjects with Parkinson’s Disease – IOS Press
Background: Reduced glucose tolerance has been long recognized as a potential risk factor for Parkinson’s disease (PD), and increasing scrutiny is currently being placed on insulin resistance (IR) as a pathologic driver of neurodegeneration. However,
for comparison, prevalence of Diabetes in the general population:
its high – 20% suggesting much higher prevalence of IR, not sure if IR in PD is that much above normal
Whole body vibration: Neuro-rehab applications
2-deoxy-D-glucose is noeroprotective
www.ncbi.nlm.nih.gov/pubmed/10398297
2-deoxy-D-glucose (2-DG; a nonmetabolizable analogue of glucose) to mice fed ad libitum. Mice receiving 2-DG exhibited reduced damage to dopaminergic neurons in the SN and improved behavioral outcome following MPTP treatment.